I called a friend whose hair I have always admired and asked her who she used. Then I called the salon and talked to the manager there about their services and personnel. Next, I made the appointment.

I was nervous before I went. Should I dress up or down? Jeans or skirt? (I finally decided on a casual skirt). I had trouble finding the place and was 20 minutes late. Everything was totally different from my old salon. This one was huge and bustling, with a large entryway/waiting room with a semicircular appointments desk big enough for three people to sit behind. First, I was introduced to my new hair colorist, then I was ushered to my new hairstylist’s chair and finally to yet a third chair where a separate person did the blow-drying because the stylist was backed up.

I approached each encounter with trepidation, but I was pleasantly surprised. The colorist couldn’t have been nicer or more professional. The hairstylist was quick and funny and gave me one of those great haircuts that you can tell works, even when it’s still wet. Even the young woman who blew-dry my hair was nice and efficient.

I felt myself settling into this new place with its 30 styling chairs, a lot of hustle and bustle and a river view. I could get used to this, I thought.

I realized at a salon so large, with so many people working there, that my cut and color was going to cost considerably more than it had at my former salon. I was right. It did.

But it was worth it. I had found a new home. It wasn’t comfy, like the old home, where I could talk gardening and fishing and family troubles with my hairdresser, who also had become my friend. There was no exchanging of book titles or favorite recipes.

On the other hand, these new folks kept asking me if I wanted something to drink, and even water came in a lovely tumbler with lots of ice and a slice of lemon with a straw. Besides, I realized it was a good place to visit, where I would come out looking a lot better than when I came in, and where everyone was welcoming and wore a smile.

I had a new bounce to my step as I left because I knew I would be coming back, but I also felt a sense of loss for my old friend, who wouldn’t be cutting my hair and sharing my joys and sorrows anymore

To prove that a person needed these nosodes, the sequential therapist points to the so-called “healing reactions” these remedies induce. Dr. Elmiger states he has given Psorinum 10M to every client, and found in many who were not psoric that the nosode still triggered the manifold symptoms of psora! Well, If you given Psorinum in 200/1M/10M to everyone on three successive days you are going to produce proving symptoms in many individuals! Is it any surprise that many of the clients undergoing this treatment would manifest “skin eruptions, bronchitis, migraines” and other such symptoms (many of which they never had before)?

Dr. Elmiger also speaks of giving Tuberculinum, Medorrhinum and Syphilinum in the same manner and getting the same “more or less vivid and explosive” reactions. Symptoms that are produced by a primary action of a potentized remedy are no proof that the person “needed” the remedy. Such an experiment only shows that homeopathic remedies are capable of producing symptoms. The symptoms caused by a primary action are signs of an improper dose, partial simillimums and wrong remedies. It is only the conservative, curative, secondary reaction of the vital force that produces true healing effects. Under the controlled direction of the vital force such healing crises are temperate, brief and complete in nature. It is by the careful application of the cardinal homeopathic principles that one attains the “rapid, gentle and permanent cure” elucidated in Aphorism 2 of The Organon.

All of these points demonstrate areas where ST differs greatly from the practice of traditional homeopathy, i.e., the lack of careful analysis of the signs and symptoms produced by homeopathic remedies both in the provings and on the sick as well as the production of unneeded homeopathic aggravations by standardized prescriptions.

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After the removal of all of the acquired blockages along the time-line, the sequential therapist begins the treatment of the inherited miasms. It is certainly a theoretical truth that everyone has all of the miasms to one degree or another. Nevertheless, to move from this assumption to the routine use of all the major nosodes on every patient is certainly a great leap. Also, at the same time, it is said in ST that there is a fixed order to the treatment of inherited miasms. First psora is treated with Psorinum, second TB with Tuberculinum, third sycosis with Medorrhinum, and fourth syphilis with Syphilinum. This seems to suppose that the inherited miasms are in some fixed order.

The concept of a fixed order of miasms is a contradiction of the principles on which ST is based, the time-line. How can there be a fixed order of the layers of the inherited miasms when they have also developed in time and space? Certainly, if a father has acquired sycosis, there is the greatest possibility that the inherited effects of the miasm will be observable in his child. This can often be seen in the very physical constitution and mental temperament of the child as well as the symptom syndromes present.

Does this child need Psorinum, then Tuberculinum, and finally Medorrhinum? Certainly not, because as the time-line extends backward through the generations, it must reflect individual mixtures of miasms not fixed program schedules given to everyone. Maybe the child needs Thuja, Natrum sulphuricum, or some other anti-sycotic remedy, rather than a nosode. The assumption that inherited miasms can only be removed by nosodes is unfounded. There are manifold case histories that prove this basic point. Exposing everyone to the exact same series of all the major nosodes has no basis in the time-line nor in the signs and symptoms of the miasmic syndromes.

Sequential therapists maintain that the basis of its fixed series of remedies can be found in Hahnemann’s writings. Although Hahnemann did give directions about the order of treatment of specific miasmic combinations in The Chronic Diseases, he did not give one set of instructions for use in all conditions. The series followed in ST has its origin in Hahnemann’s instructions on how to treat a person with a syphilitic background who contracts sycosis that is suppressed, thus activating latent psora producing a complex disease. This example can be found on the last page of Hahnemann’s discourse on Sycosis.

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Each constitution has its own unique sensitivity, which needs careful individualization of both the remedy and the treatment schedule. In Hahnemann’s advanced methodology, remedies are given exclusively in medicinal solutions rather then in a dry dose of the pills. In this way the administration of the remedy is adjusted through succussions and controlled dosages to fit the exact sensitivity of the constitution.

Such a refinement of the dose is impossible without strict individualization of treatment and the usage of the aqueous solutions of the centesimal and LM potency. These methods cause the remedies to act more gently on a deeper level, and speed the cure of complex chronic diseases without causing undue aggravations. For more information about these techniques, please read my document “Hahnemann’s Advanced Methods” in the archives of the Homeopathy Mailing List or, if necessary, contact David Little@maui.net for a copy.

Proper case management is one more reason why the maxims of the single remedy and the minimal dose are very important. How do we know which remedy is causing what effects after we have given a combination of remedies in 200, 1M and 10M on three consecutive days? There also must be some discrimination between aggravations caused by the primary action of a homeopathic remedy and healing reactions caused by the secondary action of the vital force according to Hering’s laws. Such a situation demands a deeper study of symptomatology, in which ST is very weak. This brings us to another point, the routine treatment of inherited miasms with all the major nosodes.

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In ST, it is a common practice to administer 200, 1M, and 10M on successive days. This is done with combination remedies and nosodes in a routine manner. In most cases such a dose of high potency mixtures are given once every month based on each “separate” aetiology along the time-line. Such an approach has little in common with the homeopathic doctrine of the minimal dose and the individualization of treatment. It seems there is little faith in ST in the power of the single dose and its ability to start the healing process. Many of the most striking cures are made by one single dose, although repetition is often needed in chronic diseases. In this case, however, the remedy is used only as much as is needed by the individual.

The best homeopathic cure is one in which there is rapid amelioration without any aggravations whatsoever. In ST severe, prolonged aggravations are often produced through the use of combination remedies, the isopathic use of nosodes, and triple doses of high potencies (200/1M/10M). This becomes all too obvious when one studies the cases offered in Homeopathy Renewed. All of the symptoms brought out during treatment are considered to be beneficial “healing reactions.” There is not one mention of the fact that not all symptoms that appear after the ingestion of such a series of high potency remedies are of a beneficial nature nor signs of the correct remedy.

Hahnemann taught that the “so-called homeopathic aggravation” (Aphorism 161) was caused by too strong of a primary effect of the remedy, and therefore, it is not a healing crisis. Each set of symptoms which arises after the ingestion of a homeopathic remedy must be evaluated for their peculiar nature. There is a great difference between a similar aggravation, a dis-similar aggravation and accessory symptoms brought out under treatment. Each manifestation has a different meaning and affects the future case management.

In the case of a similar aggravation there is an increase of the symptoms already present. This is a sign of the correct remedy given in the wrong dose and potency or repeated when not needed. Dis-similar aggravations are those in which there is the production of new and troublesome symptoms not appertaining to the disease being treated. This is the sign of a wrong remedy. Accessory symptoms are side-effects produced by the remedy which are un-homeopathic to the individual, and that are caused by partial simillimums. All of these signs must be carefully analyzed, as they call for different courses of action by the homeopath.

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A thin layer of cells, known as the epithelium, covers the cervix. The cells found here are either squamous (flat and scaly) or columnar (column-like). About 75 percent of cervical cancer cases come from changes in the squamous cells (squamous cell carcinoma); 15 percent to 20 percent come from changes in the squamous and columnar cells (adenosquamous carcinoma); and 5 percent to 10 percent arise from changes in just the columnar cells (adenocarcinomas) or, very rarely, in the supportive tissue around the cervix.

When the cells on the surface of the cervix become abnormal in size and shape and begin to multiply, it is a condition known as dysplasia, or preinvasive cancer. This condition occurs before the cells are considered malignant — before the cancerous cells actually invade healthy tissue. Dysplasia may or may not develop into cancer. A Pap smear will usually find these abnormal cells.

Dysplasia is classified into different categories that describe the abnormal changes in the cervix’s surface cells. The names of these categories can be confusing, as they differ depending on whether the physician is using “Pap test terms,” the “old terms” for preinvasive cancer or the “newer terms.” The chart below shows a comparison of terms:

Precancerous Conditions
Pap test results Older terms Newer terms
Questionable cell changes ASCUS/AGUS (atypical squamous/glandular cells of undetermined significance)
Early cell changes LGSIL (low-grade squamous intraepithelial lesion) Mild dysplasia or HPV infection CIN 1 (cervical intraepithelia neoplasia)
More advanced or late cell changes HGSIL (high-grade squamous intraepithelial lesion) Moderate or severe dysplasia, carcinoma in situ CIN 2 or 3 (cervical intraepithelia neoplasia)
Repeat testing in six months is the usual recommendation for an ASCUS diagnosis, while an AGUS diagnosis requires immediate investigation with colposcopy, and possibly endocervical curretage or endometrial biopsy.

In mild dysplasia, sometimes called low-grade SIL or CIN I, the cells have undergone early changes in size, shape and number. This condition may progress to cancer, but often returns to normal without treatment.

Moderate or severe dysplasia is often referred to as high-grade SIL, CIN 2 or 3, or carcinoma in situ. This condition means there are a large number of abnormal cells and they look very different from normal cells.

The cells can involve the full thickness of the epithelium, but have not yet invaded the underlying tissue. It may take up to two years or more for this condition to progress, yet it needs to be treated as soon as possible.

When the abnormal cells penetrate deeper into the cervix or spread to other tissues or organs, the disease is then called invasive cervical cancer.

The authors note in the journal that the study results “indicate that telomerase is a viable target for (anti-cancer) drugs.” Corey said. “These agents can definitely be used as a treatment of cancer.”

Corey told Reuters Health that antitelomere agents will probably not be used as a stand-alone therapy for cancer. “That’s one thing we’re pretty convinced of,” he commented.

He explained that a combination approach will probably be necessary because it takes a substantial period of time before antitelomere agents start to have an effect on cancerous cells. The approach would be to “treat the tumor with chemotherapy or surgery, and then go in with telomerase inhibitors,” Corey said. “Combining antitelomere agents with other chemotherapy agents prevents quick growth of the tumor and gives the antitelomere agents time to work,” he said.

Corey noted that the antitelomere agents seem promising for all types of cancer. “The only caveat,” he said, “is going to be concerned about what the telomere length is in a tumor.” He added, “It might be that doctors test patients for the chromosome with the shortest telomeres and then find out the average and range of telomere length in their tumor” to determine if a patient is a suitable candidate for antitelomere agents.

He pointed out that in this study, “cells with the longest telomeres showed absolutely no effect at all” from the antitelomere agents.

The authors write in the journal that antitelomerase compounds like the ones they used are already being tested in humans for conditions other than cancer. They suggest “that it should be possible to test potent antitelomerase oligonucleotides (in animals and humans) in the near future.”

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The compounds work by preventing an enzyme in cancer cells from functioning, researchers report. Cancer cells produce an enzyme telomerase that keeps telomeres from shortening, and the result is immortal cells and, ultimately, a tumor. Drugs that inhibit telomerase, therefore, should lead to reduction of cancerous tumors.

To study the effect of stopping telomerase in its tracks, Dr. B.-S. Herbert of the University of Texas Southwestern Medical Center in Dallas, and colleagues there and at the Howard Hughes Medical Institute introduced oligonucleotides. Previous studies showed that these compounds could inhibit telomerase in skin cells very similar to cancer cells.

Herbert’s group reports in the December 7th issue of Proceedings of the National Academy of Sciences that the oligonucleotide compounds inhibited telomerase, led to telomere shortening, and caused the cells to undergo cell death “with increasing frequency until no (cancer-like) cells…” remained.

They also observed that telomere shortening is reversible. This means that once the telomerase-inhibiting drugs are stopped, normal human cells will regrow their telomeres, which suggests that side effects of antitelomerase drugs might be minimal.

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Bought three potted African Violets, which I remember my grandmother growing on her windowsills.

Also visited the Web site of the African Violet Society of America, so I could learn how to help them survive and thrive. (They are still blooming!)

Scrounged around used-book sites on the Internet until I found a copy of “Auntie Mame — An Irreverent Escapade” by Patrick Dennis, the book on which the films and Broadway show were based. I love Mame — and admire her confidence and zest for life.

Decreased appetite and weight loss;
Unusual talkativeness;
Mood swings and disconnected speech;
Dilated pupils and apparent intoxication;
Sniffling, runny nose, and nosebleeds-if snorted;
Paranoia.
Health risks:

If snorted, the use may suffer holes in the cartilage separating the nostrils. Seizures and coma; Death by cardiac arrest or respiratory failure;

Suicidal behavior after prolonged use.